Lateral ganglionic intracerebral haemorrhage is most frequently due to hypertension. It includes haematomas in the putamen and the internal capsule.
Clinical features include contralateral hemiparesis, hemisensory loss, and ipsilateral conjugate eye deviation. The pupillary response is unaffected.
Large lesions produce an ipsilateral dilated or non-reactive pupil or a bilateral conjugate gaze palsy. They are associated with a poor prognosis.
Haematomas in the middle part of the putamen, affecting the the internal capsule, result in persistent hemiparesis; those towards the anterior cause less severe and less persistent motor effects; those towards the posterior may produce hemisensory loss, aphasia or visual neglect.
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