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Angiotensin II and ADH in burn injury

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As a result of hypovolaemia in a burn injury, there is decreased renal perfusion. This is sensed in the juxtaglomerular apparatus of the kidney where renin is released - see submenu. Renin ultimately results in the production of angiotensin II. Angiotensin II acts to:

  • release ADH from the hypothalamus; the latter promotes water reabsorption from the collecting ducts
  • stimulate aldosterone release from the adrenal cortex; aldosterone acts to increase water and sodium retention at the distal convoluted tubule
  • induce thirst
  • vasoconstrict the efferent renal arterioles

ADH and angiotensin II feedback to inhibit renin secretion.

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