Aetiology
- increased intake:
- excessive vitamin D
- infants on excessive amounts of high phosphate feeds
- TPN with excessive phosphate concentration
- decreased excretion:
- renal failure, with or without secondary hyperparathyroidism
- hypoparathyroidism
- redistribution:
- therapeutic destruction of cells in malignancy
- reduced celluar uptake in insulin deficiency
- acromegaly - hyperphosphataemia and hypercalcaemia may occur
- acidosis - this causes a rise in intracellular phosphate
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